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Bilaten d 16 mg (n = 11). Results of Zaditen nombre generico the study were similar to those in the previous study and results can be found in Tables S3 and S4. Discussion During the last decade, a number of studies have examined the effects of NAC consumption on the cardiovascular system. results of these studies suggest that NAC supplementation increases the synthesis of glutathione peroxidase and, consequently, decreases the activity of superoxide dismutase [23], [24]. It has also been suggested that NAC supplementation may decrease the activity of glutathione-S-transferase (GST), leading to a decrease in the formation of toxic radicals in the liver [4], [25], [26]. In addition, NAC has also been shown to enhance the recovery from effects of oxidative stress induced by and inflammatory [27], [28]. NAC is the most abundant non-protein-bound free radical scavenger in humans and can act atomoxetine 18 mg as a co-factor in the antioxidant Pharmacy online uk london defense system [4]. In addition, NAC has also been shown to act as a mitochondrial antioxidant and has been shown to be effective against the oxidative lesions induced by lipid peroxidation [29]. NAC may also be beneficial against lipid peroxidation induced by the free radical-activated protein kinase. Interestingly, NAC appears to also act as an anti-inflammatory agent [30], [31], [32]. In the generic valtrex canada pharmacy present study, we showed that a single oral dose of 15 g NAC, taken in conjunction with a low-carbohydrate, high protein diet, was able to attenuate the elevation in serum homocysteine levels induced by a high-fat diet [33]. In the absence of any significant effects on blood lipids, NAC administration appeared to improve the oxidative stress response in both the liver and adipose tissue. Our previous study showed that NAC consumption can increase serum glutathione peroxidase activity by 2-fold, which led to a decrease in the activity of superoxide dismutase (SOD). In this study, we found that a single oral dose of 15 g NAC reduced the increase in serum ferritin after a 6-hour fast [34]. Similarly, study by Goh et al. [21] reported that the administration of 5 g NAC in a high carbohydrate, low fat diet (HFF) for 4 weeks resulted in a significant decrease plasma homocysteine and increased serum glutathione. In this study, a single oral dose of NAC was able to attenuate the increase in serum homocysteine induced by a high-fat diet [7], [8]. Although these studies demonstrated a protective effects of NAC against the dietary fat, beneficial effects of NAC may be partially attributed to the increased oxidation of lipid peroxides resulting from its antioxidant properties. Indeed, NAC has been shown to inhibit the formation of lipid peroxides in both the liver and adipose tissue [7], [8], [35], [36], [37]. However, the effects of NAC on lipid peroxidation state are dependent on the type of fat consumed. In the present study, we observed a significant increase in the level of serum lipid peroxides after oral administration of 15 g NAC. The increase in lipid peroxides was not seen in the control group, and increase was significantly lower in the NAC group than placebo after a 6-hour fast. These results indicate that the NAC consumed in present study is unable to prevent the detrimental effect of a high-fat diet on the serum lipid peroxidation state. However, NAC may be able to improve the lipid peroxidation state of diet-induced hyperlipidemia and may also inhibit the increase in plasma levels of the lipid peroxides after a 6-hour fast. The mechanisms by which NAC improves the lipid peroxidation state of diet-induced hyperlipidemia are not known. It is possible that the NAC scavenging free radicals that can produce oxidative damage to lipids in the body. Thus, NAC might be able to prevent the lipid peroxidation in presence of the high-fat diet. However, it is unlikely that the increase in serum lipid peroxides is caused by NAC directly scavenging free radicals. For example, NAC has been shown to inhibit the expression of superoxide dismutase subunit SOD and the glutathione-S-transferase (GST) in liver [4], [5], [38]. A recent study reported that NAC is a strong antioxidant and that it can prevent free radical formation by blocking the activities of superoxide dismutase and glutathione-S-transferase [39]. In addition, the antioxidant effects of NAC have been reported to be mediated by the reduction of nitric oxide (NO) [40], [41].
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